Acute Low Dose Naltrexone Increases β-Endorphin and Promotes Neuronal Recovery Following Hypoxia-Ischemic Stroke in Type-2 Diabetic Mice

Keyphrases

• Opioid Growth Factor 100%
• Low-dose Naltrexone 100%
• β-endorphin 100%
• Neurological Recovery 100%
• Hypoxia-ischemia 100%
• Diabetic Mice 100%
• Ischemic Stroke 100%
• Db Mouse 80%
• Naltrexone 60%
• Infarct Size 40%
• Factor Level 40%
• Central Nervous System 40%
• Opioid Growth Factor Receptor (OGFr) 40%
• Naltrexone Treatment 40%
• Brain Tissue 40%
• Brain Regions 20%
• Clinical Outcomes 20%
• Diabetic Patients 20%
• Young Males 20%
• Opioid Receptors 20%
• Protective Effect 20%
• Autoimmune Disease 20%
• Inflammatory Cytokines 20%
• Stroke Patients 20%
• Caudate 20%
• Motor Cortex 20%
• System Response 20%
• Patient Experience 20%
• Receptor Neurons 20%
• Phosphate-buffered Saline 20%
• Immune Suppression 20%
• Neurological Deficit 20%
• Poor Recovery 20%
• Plasma Tissue 20%
• Regenerative Effect 20%
• Basic Science Study 20%

Neuroscience

• Naltrexone 100%
• Endorphin 100%
• Brain Ischemia 100%
• Growth Factor 83%
• Diabetes Mellitus 50%
• Growth Factor Receptor 33%
• Central Nervous System 33%
• Hippocampus 16%
• Autoimmune Disease 16%
• Motor Cortex 16%