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Cholestasis induces reversible accumulation of periplakin in mouse liver
Shinji Ito
, Junko Satoh
, Tsutomu Matsubara
, Yatrik M. Shah
, Sung hoon Ahn
, Cherie R. Anderson
, Weiwei Shan
,
Jeffrey M. Peters
, Frank J. Gonzalez
Veterinary and Biomedical Sciences
Penn State Cancer Institute
Huck Institutes of the Life Sciences
Cancer Institute, Mechanisms of Carcinogenesis
Research output
:
Contribution to journal
›
Article
›
peer-review
5
Scopus citations
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Keyphrases
Mouse Liver
100%
Cholestasis
100%
Periplakin
100%
Quantitative PCR
7%
Liver
7%
Physiological Role
7%
Cell Adhesion
7%
Epithelial Cells
7%
Mouse Model
7%
Etiology
7%
Wild Mice
7%
Hepatic Expression
7%
Immunohistochemistry
7%
Hepatocytes
7%
Novel Mechanism
7%
Renal Disease
7%
Western Blotting
7%
Cultured Cells
7%
Rod-like
7%
Hepatic Disease
7%
Cellular Protection
7%
Renal Tubule
7%
Junctional Complex
7%
Farnesoid X Receptor
7%
Bile Acid Receptor
7%
Epithelial Tissue
7%
Cornified Envelope
7%
Cutaneous Tissues
7%
Hepatic Accumulation
7%
Stasis
7%
Ureteral Obstruction
7%
Cytoskeletal Filaments
7%
Cytolinker
7%
Cytolinker Proteins
7%
Pharmacology, Toxicology and Pharmaceutical Science
Mouse
100%
Cholestasis
100%
Receptor
7%
Wild Type Mouse
7%
Mouse Model
7%
Western Blot
7%
Kidney Disease
7%
Bile Acid
7%
Liver Disease
7%
Ureter Obstruction
7%
Farnesoid X Receptor
7%
Biochemistry, Genetics and Molecular Biology
Periplakin
100%
Farnesoid X Receptor
7%
Bile Acid
7%