Electronic Cigarette Exposure Enhances Lung Inflammatory and Fibrotic Responses in COPD Mice

Keyphrases

• Lung 100%
• Inflammatory Response 100%
• Electronic Cigarettes 100%
• Fibrotic Response 100%
• Cigarette Exposure 100%
• Electronic Nicotine Delivery Systems 100%
• Bronchoalveolar Lavage Fluid 30%
• Overexpressing 20%
• Epithelial Sodium Channel (ENaC) 20%
• MMP-12 20%
• M2 Macrophages 20%
• Preclinical Data 10%
• TGF-1 10%
• Granulocyte Colony-stimulating Factor (G-CSF) 10%
• Macrophages 10%
• Interleukin-10 10%
• Neutrophils 10%
• Smokers 10%
• Pathological Features 10%
• Cytokine Production 10%
• Objective Outcome 10%
• Mucus Secretion 10%
• Mucus Production 10%
• Fibrogenesis 10%
• Hydroxyproline Assay 10%
• Macrophage Phenotype 10%
• COPD Patients 10%
• Subjective Outcome 10%
• IL-1R 10%
• Mucin 5AC (MUC5AC) 10%
• RAW264.7 10%
• Traditional Cigarette 10%
• Macrophage Cell Line 10%

Immunology and Microbiology

• Bronchoalveolar Lavage Fluid 100%
• M2 Macrophages 100%
• Inflammation Response 66%
• Macrophage 33%
• Interleukin 10 33%
• Neutrophil 33%
• Cytokine Production 33%
• Macrophage Colony-Stimulating Factor 33%
• Mucus Secretion 33%
• Fibrogenesis 33%
• Macrophage Cell Line 33%