Epistatic control of human obesity as revealed by linkage disequilibrium mapping: A report from the NHLBI-sponsored WISE study

Keyphrases

• Ischemia 100%
• NHLBI 100%
• Epistasis 100%
• Quantitative Trait Loci 100%
• Linkage Disequilibrium Mapping 100%
• Obesity 75%
• Single nucleotide Polymorphism 50%
• Body Mass Index 25%
• Receptor-interacting Protein Kinase 1 (RIPK1) 25%
• Diet Exercise 25%
• Epistatic Effect 25%
• Genetic Architecture 25%
• G Protein 25%
• White Women 25%
• Africa 25%
• Cardiovascular Disease 25%
• Hypertension 25%
• Linkage Disequilibrium Analysis 25%
• Black Population 25%
• Association Study 25%
• Adrenergic Receptors 25%
• Black Women 25%
• Alpha Subunit 25%
• Population Differentiation 25%
• Evolution Mechanism 25%
• Thermogenesis 25%
• Major Risk Factors 25%
• Lipolysis 25%
• White Population 25%
• Obesity Genes 25%
• Human Adipose Tissue 25%
• Type 2 Diabetes Risk 25%
• Gene Age 25%

Pharmacology, Toxicology and Pharmaceutical Science

• Ischemia 100%
• Syndrome 100%
• Malignant Neoplasm 50%
• Receptor Protein 50%
• Disease 50%
• Non Insulin Dependent Diabetes Mellitus 50%
• Cardiovascular Disease 50%
• Adrenergic Receptor 50%
• Stimulatory Guanine Nucleotide Binding Protein 50%

Biochemistry, Genetics and Molecular Biology

• Gene Linkage Disequilibrium 100%
• Quantitative Trait Locus 100%
• Candidate Gene 50%
• Single-Nucleotide Polymorphism 50%
• Body Weight 50%
• Genetic Architecture 25%
• Body Mass 25%
• Adrenergic Receptor 25%
• Alpha Chain 25%
• Thermogenesis 25%
• Population Differentiation 25%
• Lipolysis 25%