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ESE3 inhibits pancreatic cancer metastasis by upregulating E-cadherin
Tiansuo Zhao
, Wenna Jiang
, Xiuchao Wang
, Hongwei Wang
, Chen Zheng
, Yang Li
, Yan Sun
, Chongbiao Huang
, Zhi Bo Han
,
Shengyu Yang
, Zhiliang Jia
, Keping Xie
, He Ren
, Jihui Hao
Penn State Cancer Institute
Cancer Institute, Mechanisms of Carcinogenesis
Department of Cell and Biological Systems
Research output
:
Contribution to journal
›
Article
›
peer-review
50
Scopus citations
Overview
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Keyphrases
Adenocarcinoma Cells
25%
Adenocarcinoma Metastases
25%
Cell Invasiveness
25%
Cell Motility
25%
Development Program
25%
E-cadherin
100%
E-cadherin Expression
25%
Epithelial Tissue
25%
Human Pancreatic Ductal Adenocarcinoma
25%
Lymph Node Metastasis
25%
Mechanistic Studies
25%
Metastasis
50%
Negative Regulator
25%
Orthotopic Model
50%
Overall Survival
25%
Pancreatic Cancer
25%
Pancreatic Duct Cells
50%
Pancreatic Ductal Adenocarcinoma
100%
Pancreatic Tissue
25%
Reduced Expression
25%
Tumor Suppressor
25%
Medicine and Dentistry
Cell Migration
14%
Cell Motility
14%
Epithelium
14%
Lymph Node Metastasis
14%
Metastatic Carcinoma
100%
Overall Survival
14%
Pancreas Adenocarcinoma
100%
Pancreas Cancer
100%
Transcription Factors
14%
Tumor Suppressor Protein
14%
Upregulation
14%
Uvomorulin
100%
Immunology and Microbiology
Adenocarcinoma Cell Line
33%
Cadherin
100%
Cell Migration
33%
Cell Motility
33%
Lymph Node
33%
Mouse Model
66%
Overall Survival
33%
Transcription Factors
33%
Upregulation
33%
Pharmacology, Toxicology and Pharmaceutical Science
Lymph Node Metastasis
14%
Mouse Model
28%
Overall Survival
14%
Pancreas Adenocarcinoma
100%
Pancreas Cancer
100%
Transcription Factors
14%
Tumor Suppressor Protein
14%
Uvomorulin
100%
Biochemistry, Genetics and Molecular Biology
Cadherin
100%
Cell Migration
33%
Cell Motility
33%
Mouse Model
66%
Overall Survival
33%
Transcription
33%
Tumor Suppressor Protein
33%
Upregulation
33%