Abstract
• An infant with Hirschsprung’s enterocolitis developed a fulminant secretory diarrhea unresponsive to all conventional therapy until cholestyramine was administered. A 12-fold decrease in prostaglandin E (PGE) levels in the colostomy fluid was documented in response to cholestyramine therapy. It is postulated that increased PGE activity, errterotoxin, and bile acid malabsorption may be involved in the enterocolitis of Hirschsprung's disease.
| Original language | English (US) |
|---|---|
| Pages (from-to) | 417-418 |
| Number of pages | 2 |
| Journal | Journal of pediatric surgery |
| Volume | 13 |
| Issue number | 4 |
| DOIs | |
| State | Published - 1978 |
UN SDGs
This output contributes to the following UN Sustainable Development Goals (SDGs)
-
SDG 3 Good Health and Well-being
All Science Journal Classification (ASJC) codes
- Surgery
- Pediatrics, Perinatology, and Child Health
Fingerprint
Dive into the research topics of 'Hirschsprung's enterocolitis, prostaglandins, and response to cholestyramine'. Together they form a unique fingerprint.Cite this
- APA
- Author
- BIBTEX
- Harvard
- Standard
- RIS
- Vancouver