Human papillomavirus deregulates the response of a cellular network comprising of chemotactic and proinflammatory genes

Keyphrases

• Keratinocytes 100%
• High-risk Human Papillomavirus (HR-HPV) 100%
• Cellular Networks 100%
• Inflammatory Genes 100%
• HPV16 66%
• Interleukin-1β 33%
• Non-self 33%
• Evade 33%
• Innate Immune Response 33%
• Host Immunity 33%
• Immune System 33%
• Inflammatory Cytokines 33%
• Pregenomic RNA (pgRNA) 33%
• Infected Cells 33%
• Inflammatory Response 33%
• Virus Persistence 33%
• Proviral DNA 33%
• Deregulation 33%
• Latent Infection 33%
• Human Keratinocytes (HaCaT) 33%
• Downstream Signaling 33%
• Genome-wide Expression 33%
• Expression Profiling 33%
• Poly IC 33%
• Antigen-presenting 33%
• Pathogen Recognition Receptors 33%
• Protein-protein Interaction Network Analysis 33%
• Adaptive Immunity 33%
• HPV Infection 33%
• Episomal 33%
• RNA Polymerase III 33%
• Toll-like Receptor 3 (TLR3) 33%
• Retinoic Acid-inducible Gene I (RIG-I) 33%
• Intracellular Pathogen 33%
• Melanoma Differentiation-associated Gene 5 (MDA5) 33%
• Inflammasome 33%

Immunology and Microbiology

• Keratinocyte 100%
• Wart Virus 100%
• Infectious Agent 50%
• Human Papillomavirus Type 16 50%
• Virus 50%
• Protein Interaction 25%
• Adaptive Immune System 25%
• Innate Immune System 25%
• Proinflammatory Cytokine 25%
• Inflammation Response 25%
• Cytokine 25%
• RNA Polymerase 25%
• Interleukin-1 25%
• Downregulation 25%
• Polyinosinic:polycytidylic Acid 25%
• TLR3 25%
• Chemokine 25%
• RIG-I 25%
• Immune System 25%
• Inflammasome 25%