Loss of Surfactant Protein A Alters Perinatal Lung Morphology and Susceptibility to Hyperoxia-Induced Bronchopulmonary Dysplasia

Keyphrases

• Surfactant Protein A (SP-A) 100%
• Hyperoxia 100%
• Bronchopulmonary Dysplasia 100%
• Lung 27%
• Oxidative Damage 27%
• Alveolar Formation 27%
• Lung Tissue 18%
• Lung Macrophages 18%
• Normoxia 18%
• Radial Alveolar Count 18%
• Mean Linear Intercept 18%
• Alveolar Development 18%
• Fold Increase 9%
• Genetic Variants 9%
• 4-fold 9%
• Mouse Model 9%
• Macrophages 9%
• Aged Mice 9%
• Clinically Significant 9%
• Surfactant 9%
• Late Gestation 9%
• Alveolar Macrophages 9%
• Temporal Interaction 9%
• Lung Development 9%
• Normoxic 9%
• Neonatal Lung Disease 9%
• Morphometric Analysis 9%
• Parturition 9%
• Proteomic Profiling 9%
• Alveolarization 9%
• Preterm Lung 9%
• DNA Damage Markers 9%
• WT Mice 9%
• Spatial Proteomics 9%

Medicine and Dentistry

• Surfactant Protein A 100%
• Lung Dysplasia 100%
• Hyperoxia 100%
• Lung 100%
• Macrophage 27%
• Oxidizing Agent 18%
• Breathing 9%
• Silo-Filler's Disease 9%
• Pregnancy 9%
• DNA Damage 9%
• Alveolar Macrophage 9%
• Neonatal Infant 9%
• Lung Development 9%
• Alveolarization 9%
• Proteomics 9%
• Morphometric Analysis 9%