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Mono-ADP-ribosylation by PARP10 and PARP14 in genome stability
Ashna Dhoonmoon,
Claudia M. Nicolae
Penn State Cancer Institute
Cancer Institute, Next-Generation Therapies
Department of Molecular and Precision Medicine
Research output
:
Contribution to journal
›
Article
›
peer-review
12
Scopus citations
Overview
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Dive into the research topics of 'Mono-ADP-ribosylation by PARP10 and PARP14 in genome stability'. Together they form a unique fingerprint.
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Keyphrases
Genome Stability
100%
PARP10
100%
PARP14
100%
Mono-ADP-ribosylation
100%
Poly(ADP-ribose) Polymerase
50%
Post-translational Modification
33%
Cellular Processes
16%
In Cancer
16%
Cell Proliferation
16%
DNA Repair
16%
Cancer Progression
16%
Transcriptional Regulation
16%
Therapeutic Target
16%
Oncogenesis
16%
PARP Inhibitor (PARPi)
16%
DNA Damage Repair
16%
ADP-ribosylation
16%
Poly(ADP-ribose)
16%
Anticancer Immune Response
16%
ADP-ribosyltransferase
16%
Biochemistry, Genetics and Molecular Biology
Adenosine Diphosphate
100%
Genome Instability
100%
Poly ADP Ribose Polymerase
100%
Posttranslational Modification
66%
DNA Repair
66%
Enzyme
33%
Cell Proliferation
33%
Carcinogenesis
33%
PARP1
33%
PARP2
33%
Immunology and Microbiology
Genomic Instability
100%
Adenosine Diphosphate Ribosylation
100%
DNA Repair
66%
Post-Translational Modification
66%
Cell Proliferation
33%
Pharmacology, Toxicology and Pharmaceutical Science
Adenosine Diphosphate
100%
Nicotinamide Adenine Dinucleotide Adenosine Diphosphate Ribosyltransferase
33%
Malignant Neoplasm
11%
Carcinogenesis
11%
Cancer Growth
11%
Cellular Processes
11%