NLRX1 modulates immunometabolic mechanisms controlling the Host-Gut microbiota interactions during inflammatory bowel disease

Immunology and Microbiology

• Inflammatory Bowel Disease 100%
• Intestine Flora 100%
• Microbiome 100%
• Cell Metabolism 80%
• Gut Microbiome 80%
• Wild Type Mouse 60%
• Cell Function 60%
• Wild Type 40%
• Glutamine 40%
• Decision Support System 20%
• Immune Response 20%
• Colitis 20%
• Cell Specificity 20%
• Taxon 20%
• Cytokine Response 20%
• Tight Junction 20%
• Heredity 20%
• Amino Acid Metabolism 20%
• Organoid 20%
• Chemokine Response 20%
• Veillonella 20%

Keyphrases

• Inflammatory Bowel Disease 100%
• Immunometabolic 100%
• Gut Microbiota Interaction 100%
• Intestinal Epithelial Cells 33%
• Cell Metabolism 26%
• Gut Microbiome 26%
• Microbiome 20%
• Wild Mice 20%
• Disease Severity 20%
• Inflammation 13%
• Epithelial Cells 13%
• Cell Function 13%
• Microbe-microbe Interactions 13%
• Controller 6%
• Metabolism 6%
• Glutamine 6%
• Genetic Factors 6%
• Defense Response 6%
• Whole-body 6%
• Inflammatory Markers 6%
• Cell-type Specificity 6%
• Mediating Effect 6%
• Cell Behavior 6%
• Metabolic Profiling 6%
• Tight Junction 6%
• Cytokine Expression 6%
• Specificity Studies 6%
• Response Factor 6%
• Antimicrobial Defense 6%
• Chemokine Expression 6%
• Amino Acid Metabolism 6%
• Bacterial Taxa 6%
• Sirtuin 1 (SIRT1) 6%
• Barrier Permeability 6%
• Glutamine Supplementation 6%
• Veillonella 6%
• Cre-loxP 6%
• Microbiological Analysis 6%
• Mucosal Inflammation 6%
• Colonic Lesions 6%
• Dysregulated Immune Response 6%
• Immune Genetic 6%