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Pathogenic mutations perturb calmodulin regulation of Na
v
1.8 channel
Liang Hong
, Meihong Zhang
, Arvind Sridhar
,
Dawood Darbar
Department of Medicine
Penn State Heart and Vascular Institute
Research output
:
Contribution to journal
›
Article
›
peer-review
12
Link opens in a new tab
Scopus citations
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Dive into the research topics of 'Pathogenic mutations perturb calmodulin regulation of Na
v
1.8 channel'. Together they form a unique fingerprint.
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Keyphrases
Calmodulin
100%
Pathogenic mutations
100%
NaV1.8 Channels
100%
Brugada Syndrome
37%
Atrial Fibrillation
37%
Slow Inactivation
37%
IQ Domain
37%
Channel Function
25%
Voltage-gated Sodium Channel
25%
Arrhythmia
12%
Hyperpolarized
12%
Channel Gating
12%
Cardiac Action Potential
12%
Ventricular Arrhythmia
12%
Hyperpolarization
12%
Sodium Current
12%
Inactivation Curve
12%
Nav1.8
12%
Cardiac Conduction
12%
Steady-state Inactivation
12%
SCN10A
12%
Calmodulin-binding
12%
Biochemistry, Genetics and Molecular Biology
Nav1.8
100%
Calmodulin
100%
Brugada Syndrome
33%
Atrial Fibrillation
33%
Sodium Channel
22%
Electric Potential
22%
Cardiac Dysrhythmia
22%
Steady State
11%
Channel Gating
11%
Cardiac Action Potential
11%
Sodium Current
11%
Heart Conduction
11%
Neuroscience
Sodium Channel Nav1.8
100%
Calmodulin
100%
Atrial Fibrillation
33%
Brugada Syndrome
33%
Voltage Gated Sodium Channel
22%
Cardiac Dysrhythmia
22%
Channel Gating
11%
Cardiac Action Potential
11%
Heart Conduction
11%
Sodium Current
11%
SCN10A
11%