Senescence-accelerated mice (SAMs) as a model for brain aging and immunosenescence

Keyphrases

• Age-related 25%
• Age-related Changes 25%
• Age-related Decline 25%
• Age-related Illnesses 50%
• Antigen-presenting Cells 25%
• Autoantibodies 25%
• Brain Aging 100%
• Brain Atrophy 25%
• Brain Cells 25%
• Brain Tissue 25%
• CD4+ T Cells 25%
• Cholinergic Receptors 25%
• Cortical Neurons 25%
• Depressive-like Behavior 25%
• DNA Damage 25%
• Dysregulation 25%
• Early Onset 25%
• Forming Capacity 25%
• Human Aging 25%
• Impaired Memory 25%
• Inbred Strains 25%
• Inflammatory Cytokines 25%
• Mouse Brain 25%
• Natural Killer Activity 25%
• Neuronal Dendrites 25%
• Neurotrophic Factors 25%
• SAMP10 100%
• Senescence 50%
• Senescence-accelerated Mouse 100%
• Synapse 25%
• T Cell Activation 25%
• T Helper Cells 25%
• Tissue Homeostasis 25%
• Virus Infection 25%
• Zinc 25%
• Zinc Transporter 25%

Neuroscience

• Antigen Presenting Cell 50%
• Autoantibodies 50%
• Behavior (Neuroscience) 50%
• CD4 50%
• Cell Function 50%
• Cholinergic Receptor 50%
• Dendrite 50%
• Dendritic Spine 50%
• DNA Damage 50%
• Learning Disabilities 50%
• Natural Killer Cell 50%
• Neurodegeneration 50%
• Neurotrophic Factor 50%
• Proinflammatory Cytokine 50%
• Synapse 50%
• T Cell 100%
• Zinc Transporter 50%