Signaling Dependent and Independent Mechanisms in Pemphigus Vulgaris Blister Formation

Keyphrases

• Pemphigus Vulgaris 100%
• Desmoglein 3 100%
• Blister Formation 100%
• Pemphigus 50%
• Endocytosis 25%
• P38 Mitogen-activated Protein Kinase (p38 MAPK) 25%
• Monoclonal Antibody 12%
• Adhesion 12%
• MAPK Inhibitors 12%
• Cell Adhesion 12%
• Tyrosine Kinase 12%
• Pathogenicity 12%
• Cell Surface 12%
• Epidermis 12%
• Keratinocyte Cells 12%
• Pathogenic Mechanism 12%
• Autoantibodies 12%
• Autoimmune 12%
• Cell Adhesion Strength 12%
• Mouse Monoclonal Antibody 12%
• Pathomechanism 12%
• Blistering Disease 12%
• Systems Intervention 12%
• Desmosomal Cadherins 12%
• Pathogenic Activity 12%
• Therapeutic Interventions 12%
• Keratinocyte Adhesion 12%

Immunology and Microbiology

• Pemphigus vulgaris 100%
• Pemphigus 57%
• Desmoglein 3 Antibody 57%
• Monoclonal Antibody 28%
• Keratinocyte 28%
• Endocytosis 28%
• Pathogenicity 14%
• Cell Surface 14%
• Cell Adhesion 14%
• Cadherin 14%
• Autoimmunity 14%
• Autoantibodies 14%
• Explant 14%
• Tyrosine 14%
• In Vitro 14%
• Phosphotransferase 14%
• Kinase 14%

Biochemistry, Genetics and Molecular Biology

• Desmoglein-3 100%
• Keratinocyte 25%
• Endocytosis 25%
• Monoclonal Antibody 25%
• P38 Mitogen-Activated Protein Kinases 25%
• Cell Adhesion 12%
• Explant 12%
• Receptor Tyrosine Kinase 12%
• Tyrosine Kinase 12%
• Autoantibodies 12%
• Autoimmunity 12%
• Cadherin 12%