Abstract
We describe a neonate that developed alternating wide and narrow complex tachycardias following heart surgery for congenital heart defect. Although the etiology was sinus tachycardia, a premature atrial complex initiated aberrancy due to phase 3 block that was perpetuated due to ‘linking’ at similar or even slower heart rates. Similarly, there was abrupt transition from wide to narrow complex tachycardia following a premature ventricular complex without a change in subsequent heart rate. This was explained by interruption of linking phenomenon by premature ventricular activation. While the above mechanisms are described in the setting of a supraventricular tachyarrhythmia, this has not been reported during sinus tachycardia.
Original language | English (US) |
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Pages (from-to) | 357-361 |
Number of pages | 5 |
Journal | Journal of Electrocardiology |
Volume | 51 |
Issue number | 3 |
DOIs | |
State | Published - May 1 2018 |
All Science Journal Classification (ASJC) codes
- Cardiology and Cardiovascular Medicine