Skip to main navigation
Skip to search
Skip to main content
Penn State Home
Help & FAQ
Home
Researchers
Research output
Research units
Equipment
Grants & Projects
Prizes
Activities
Search by expertise, name or affiliation
The O
6
-methylation of 4-hydroxyestradiol is inhibited by 2-hydroxyestradiol: Implications for estrogen-induced carcinogenesis
Deodutta Roy
, Judith Weisz
, Joachim G. Liehr
Department of Obstetrics and Gynecology
Research output
:
Contribution to journal
›
Article
›
peer-review
65
Scopus citations
Overview
Fingerprint
Fingerprint
Dive into the research topics of 'The O
6
-methylation of 4-hydroxyestradiol is inhibited by 2-hydroxyestradiol: Implications for estrogen-induced carcinogenesis'. Together they form a unique fingerprint.
Sort by
Weight
Alphabetically
Keyphrases
Estrogen
100%
Methylation
100%
Oncogenesis
100%
O-methylation
100%
4-hydroxyestradiol
100%
2-methoxyestradiol
100%
Vmax
40%
Catechol Estrogens
40%
Inhibition Constant
40%
Methyl Ethers
40%
Kidney
20%
Hamster
20%
Optimum pH
20%
DNA Damage
20%
Apparent Affinity
20%
Metabolic Clearance Rate
20%
Redox Cycling
20%
Rat pituitary
20%
Catechol-O-methyltransferase
20%
Mixed Inhibition
20%
Pharmacology, Toxicology and Pharmaceutical Science
Carcinogenesis
100%
2 Hydroxyestradiol
100%
Catechol Estrogen
66%
Mouse
33%
Catechol
33%
Ether Derivative
33%
Dimethyl Ether
33%
Methyltransferase
33%
Intrinsic Clearance
33%
Chemistry
Methylation
100%
% Inhibition
28%
Metabolic
28%
Deoxyribonucleic Acid
14%
Catechol
14%
Carcinogenic
14%
Km
14%