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TRP channels as mediators of oxidative stress
Barbara A. Miller
, Wenyi Zhang
Department of Pediatrics
Division of Hematology and Oncology
Research output
:
Chapter in Book/Report/Conference proceeding
›
Conference contribution
103
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Scopus citations
Overview
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Dive into the research topics of 'TRP channels as mediators of oxidative stress'. Together they form a unique fingerprint.
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Keyphrases
Oxidative Stress
100%
TRP Channels
100%
Cell Proliferation
66%
Cell Death
66%
TRPM7
66%
Transient Receptor Potential Melastatin 2 (TRPM2)
66%
Oxidant Stress
66%
Ischemia-reperfusion Injury
33%
Protein Family
33%
Family Members
33%
Induced Cell Death
33%
Cell Survival
33%
Overexpression
33%
Physiologic
33%
Diabetes
33%
Mammalian Cells
33%
Oxidative Damage
33%
Ion Channels
33%
Chemical Stimulation
33%
Therapeutic Approaches
33%
Ca2+ Influx
33%
TRPC4
33%
Diverse Groups
33%
B Cells
33%
TRPC3
33%
Mechanical Stimulation
33%
Cortical Neurons
33%
Sequence Identity
33%
Human Aortic Endothelial Cells
33%
Diabetic Sensory Neuropathy
33%
Neuronal Injury
33%
Cell Systems
33%
Transient Receptor Potential Vanilloid 1 (TRPV1)
33%
Tissue Damage
33%
Additional Channel
33%
Permeable Channel
33%
Amyloid-β Peptide
33%
Transient Receptor Potential Protein
33%
Redox-sensitive
33%
Physical Stimuli
33%
Biochemistry, Genetics and Molecular Biology
Oxidative Stress
100%
Mediator
100%
Transient Receptor Potential Channel
100%
Cell Death
60%
Isoform
40%
Cell Proliferation
40%
TRPM7
40%
TRPM2
40%
Transient Receptor Potential Melastanin
40%
Reperfusion
20%
Cell Survival
20%
Tumor Necrosis Factor
20%
Chelation
20%
Receptor Potential
20%
TRPC3
20%
TRPC4
20%
B Cell
20%
Transient Receptor Potential Vanilloid
20%
Protein Superfamily
20%
TRPV1
20%
Ion Channel
20%
Amyloid Protein
20%
Peptide
20%